![]() Reflecting such versatile functions of JNK in signal transduction, a growing body of evidence now suggests that JNKs may also play a significant role in the biology of human cancers ( 3– 5). ![]() ![]() The c-Jun NH 2-terminal kinases (JNKs) belong to a subfamily of mitogen-activated protein kinases implicated in the transduction of a wide array of intracellular signals associated with, for instance, cellular proliferation, differentiation, development, the inflammatory response and apoptosis ( 1, 2).
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